Antibiotic treatment does not reduce the risk of secondary cardiac events, the study shows
Taking antibiotics weekly for 1 year does not reduce the risk of heart attack or other cardiac events in patients with stable coronary heart disease, according to a study by the University of Washington. The study was 22nd in the April New England Journal of Medicine. Previous studies have Chlamydia pneumoniae, the bacteria found in arterial plaque in patients with coronary heart disease. Some doctors felt that the removal of C.Events. The prescription of antibiotics for this purpose will not be tested by a randomized clinical trial. The questioning by the fact that antibiotics used to treat bacteria and reduce the risk of cardiac events was conducted in 27 different locations in the United States. All adults in the United States have been exposed to C. pneumoniae, which
spreads through the air and causes pneumonia or mild respiratory illness at some point in their lives. This study, to study coronary events called azithromycin, or ACES, found no benefit with treatment C.with an antibiotic to reduce the risk of heart attacks and improve overall cardiac outcomes. ACES researchers randomized 4012 men and women who either dose of azithromycin once weekly or placebo for a year, from receipt in 1999. After a median follow-up of 3.9 years, there was a significant reduction of cardiac events, including death, myocardial infarction, unstable angina, angioplasty or heart surgery, defined by participants receive antibiotics compared with those taking placebo .
tobacco and snuff, or the presence of C. pneumoniae antibodies. Antibiotic treatment
had no effect on total mortality and the incidence of stroke. Men and women were included in the study if they had stable coronary artery disease after a previous cardiac event such as a heart attack, angioplasty or cardiac bypass. Azithromycin was selected for its proven effectiveness against bacteria C. pneumoniae and the dose once a week.was not developed for the role of C. pneumoniae in the pathogenesis of
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